journal of pharmacological sciences سال:2010 - دوره:112

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journal of pharmacological sciences
سال:2010 - دوره:112 - شماره:2

A novel CC-chemokine receptor 3 antagonist,Ki19003,inhibits airway eosinophilia and subepithelial/peribronchial fibrosis induced by repeated antigen challenge in mice - صفحه:203-213 Co-operative effect between γ-aminobutyric acid A receptors and central-type benzodiazepine receptors on amylase release in rat parotid acinar cells - صفحه:247-250 Demonstration of direct neurite-osteoclastic cell communication in vitro via the adrenergic receptor - صفحه:184-191 Effect of diesel exhaust particles on house dust mite-induced airway eosinophilic inflammation and remodeling in mice - صفحه:192-202 Effect of erythropoietin on angiogenesis with the increased adhesion of platelets to the microvessels in the hind-limb ischemia model in mice - صفحه:167-175 Ginsenoside-Rb1 attenuates dilated cardiomyopathy in cTnTR141W transgenic mouse - صفحه:214-222 MMHD [(S,E)-2-methyl-1-(2-methylthiazol-4-yl) hexa-1,5-dien-ol],a novel synthetic compound derived from epothilone,suppresses nuclear factor-κB-mediated cytokine expression in lipopolysaccharide-stimulated BV-2 microglia - صفحه:158-166 Methylglyoxal enhances sodium nitroprusside-induced relaxation in rat Aorta - صفحه:176-183 Nitric oxide and carbon monoxide act as inhibitory neurotransmitters in the longitudinal muscle of C57BL/6J mouse distal colon - صفحه:231-241 Sodium-calcium exchangers in rat ameloblasts - صفحه:223-230 The forefront for novel therapeutic agents based on the pathophysiology of lower urinary tract dysfunction: Ameliorative effect of solifenacin succinate (Vesicare®),a bladder-selective antimuscarinic agent,on overactive bladder symptoms,especially urgency episodes - صفحه:135-141 The forefront for novel therapeutic agents based on the pathophysiology of lower urinary tract dysfunction: Bladder selectivity based on in vivo drug-receptor binding characteristics of antimuscarinic agents for treatment of overactive bladder - صفحه:142-150 The forefront for novel therapeutic agents based on the pathophysiology of lower urinary tract dysfunction: Pathophysiology and pharmacotherapy of overactive bladder - صفحه:128-134 The forefront for novel therapeutic agents based on the pathophysiology of lower urinary tract dysfunction: Pathophysiology of voiding dysfunction and pharmacological therapy - صفحه:121-127 The forefront for novel therapeutic agents based on the pathophysiology of lower urinary tract dysfunction: α-blockers in the treatment of male voiding dysfunction - How do they work and why do they differ in tolerability? - صفحه:151-157 The protective effect of radicicol against renal ischemia-reperfusion injury in mice - صفحه:242-246 Tumor necrosis factor-α mediates the blood-brain barrier dysfunction induced by activated microglia in mouse brain microvascular endothelial cells - صفحه:251-254