journal of pharmacological sciences سال:2010 - دوره:113

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journal of pharmacological sciences
سال:2010 - دوره:113 - شماره:1

15d-prostaglandin J2 enhancement of nerve growth factor-induced neurite outgrowth is blocked by the chemoattractant receptor-homologous molecule expressed on T-helper type 2 cells (CRTH2) antagonist CAY10471 in PC12 cells - صفحه:89-93 Ameliorative effect of a hippocampal metabotropic glutamate-receptor agonist on histamine H1 receptor antagonist-induced memory deficit in rats - صفحه:41-47 Contribution of chloride channel conductance to the regulation of resting membrane potential in chondrocytes - صفحه:94-99 Engineered mutation of some important amino acids in angiotensin II type 1 (AT1) receptor increases the binding affinity of AT1-receptor antagonists - صفحه:57-65 Glycyrrhizin attenuates mucus production by inhibition of MUC5AC mRNA expression in vivo and in vitro - صفحه:76-83 Herb formula Fufangqishe-Pill prevents upright posture-induced intervertebral disc degeneration at the lumbar in rats - صفحه:23-31 Identification of cells expressing cyclooxygenase-2 in response to interleukin-1β in rat aortae - صفحه:84-88 Increased nitrosoglutathione reductase activity in hypoxic pulmonary hypertension in mice - صفحه:32-40 Novel etiological and therapeutic strategies for neurodiseases: Epigenetic understanding of gene-environment interactions - صفحه:3-8 Novel etiological and therapeutic strategies for neurodiseases: Mechanisms and consequences of febrile seizures: Lessons from animal models - صفحه:14-22 Novel etiological and therapeutic strategies for neurodiseases: Preface - صفحه:1-2 Novel etiological and therapeutic strategies for neurodiseases: RNA editing enzyme abnormality in sporadic amyotrophic lateral sclerosis - صفحه:9-13 Silibinin induced autophagic and apoptotic cell death in HT1080 cells through a reactive oxygen species pathway - صفحه:48-56 Tamoxifen inhibits cardiac ATP-sensitive and acetylcholine-activated K+ currents in part by interfering with phosphatidylinositol 4,5-bisphosphate-channel interaction - صفحه:66-75