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journal of dental research
  
سال:2017 - دوره:96 - شماره:11
  
 
A Population-Based Study of Effects of Genetic Loci on Orofacial Clefts
- صفحه:1322-1329
  
 
ARHGAP29 Mutation Is Associated with Abnormal Oral Epithelial Adhesions
- صفحه:1298-1305
  
 
Abnormal WNT5A Signaling Causes Mandibular Hypoplasia in Robinow Syndrome
- صفحه:1265-1272
  
 
Anti-EDAR Agonist Antibody Therapy Resolves Palate Defects in Pax9-/- Mice
- صفحه:1282-1289
  
 
Candidate Genes for Nonsyndromic Cleft Palate Detected by Exome Sequencing
- صفحه:1314-1321
  
 
Closing the Gap: Mouse Models to Study Adhesion in Secondary Palatogenesis
- صفحه:1210-1220
  
 
Disrupted IRF6-NME1/2 Complexes as a Cause of Cleft Lip/Palate
- صفحه:1330-1338
  
 
Face Forward: Gene Variants,Pathways,and Therapies for Craniofacial Anomalies
- صفحه:1181-1183
  
 
Grainyhead-like Transcription Factors in Craniofacial Development
- صفحه:1200-1209
  
 
Hippo Pathway: An Emerging Regulator of Craniofacial and Dental Development
- صفحه:1229-1237
  
 
IRF6 and SPRY4 Signaling Interact in Periderm Development
- صفحه:1306-1313
  
 
Inhibition of the MIR-17-92 Cluster Separates Stages of Palatogenesis
- صفحه:1257-1264
  
 
Modulating Wnt Signaling Rescues Palate Morphogenesis in Pax9 Mutant Mice
- صفحه:1273-1281
  
 
Molecular and Cellular Mechanisms of Palate Development
- صفحه:1184-1191
  
 
Signaling Pathways Critical for Tooth Root Formation
- صفحه:1221-1228
  
 
The Biology Underlying Abnormalities of Tooth Number in Humans
- صفحه:1248-1256
  
 
The Impact of the Eda Pathway on Tooth Root Development
- صفحه:1290-1297
  
 
The Role of Noncoding Genetic Variation in Isolated Orofacial Clefts
- صفحه:1238-1247
  
 
The Use of Variant Maps to Explore Domain-Specific Mutations of FGFR1
- صفحه:1339-1345
  
 
Using Zebrafish to Test the Genetic Basis of Human Craniofacial Diseases
- صفحه:1192-1199
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