Myocardial Infarction causing remodeling of cardiomyoctyes

Aim: to observe immediate changes in cardiomyocytes repair after myocardial infarction and it's possible treatment in tertiary care hospitals of lahore. methods: in this observational study 1250 patients (mean 48±5 years,58% males) with myocardial infarction after 4 weeks. infarcted marks in the myocardium cell enhances receptor signaling,while commencement of releasing oxygen provoke cytokine and chemokine. high population of invading leucocytes remove the infarct from dead cell area,on the other hand give boost to cells that escort to making a scar. results: treatment of exact inflammatory agents through medication may not reducing the quantity of cardiomyocytes in the infracted area,but can shield the heart chamber from tissue dilatation and remodeling,which may leads to heart failure after myocardial infarction. the malfunction of medication in patients with myocardial infarction may lead to surgical intervention. conclusion: treatment in response to inflammatory response to ease unconditional remodeling of cardiomyocytes in patients with myocardial infarction in quiet helpful and having good prognosis .patients,which shows aggressive remodeling may achieve formation of new cardiac muscles.